TASS, June 22. People who are treated in intensive care with severe COVID-19, is about ten times more likely to suffer from arrhythmia and related disorders in heart function than patients with mild or moderate forms of coronavirus infection. To such conclusion scientists, article which was published the scientific journal Heart Rhythm.
“To protect patients it is important to understand how COVID-19 affects different organs. Our observations show that nocardioides problems, such as systemic inflammation and spread to other parts of the body, affect the probability of heart failure and the development of arrhythmias more than direct damage to the cardiac tissue by the virus,” said one of the study’s authors, associate Professor, University of Pennsylvania (USA), Rajat Deo.
this feature of the virus, as scientists now suggest, may explain why many cases COVID-19 suffer not only from disorders in the respiratory system, but also lose their sense of smell and also have problems with digestion and malfunctions of the circulatory system.
Deo and his colleagues discovered another cardiac manifestation of coronavirus infection. They tracked the changes in the health status of approximately seven hundred patients who came to University hospital with a diagnosis of COVID-19 between early March and mid-may. Some of them suffered from light to medium forms of infection and other doctors was transferred to the intensive care unit.
the authors of the article analyzed the history of their disease and revealed an unusual trend — patients with the most severe forms COVID-19 much more often than other patients suffered from arrhythmia and cardiac arrest.
In total, the University hospital with coronavirus-infected patients occurred 53 arrhythmia different forms, as well as nine cardiac arrests. While none of the patients with COVID-19 or moderate forms of gravity your heart going.
Scientists found no serious differences in the condition of the cardiac tissue in patients from the other group. This may indicate that the heart problems were not associated with the penetration of SARS-CoV-2 in the muscle cells and how the virus affects immune function.
While scientists don’t know how permanent such changes in the circulatory system. As I hope Tao and his colleagues, follow-up and experiments with various drugs, including anticoagulants, will help to understand how to protect patients from the long-term consequences of infection with coronavirus.
